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Considerando-se os tipos de frases em Libras, analisar os itens.
I. Na forma afirmativa, a expressão facial é neutra.
II. Na forma exclamativa, as sobrancelhas são franzidas e há um ligeiro movimento da cabeça inclinando-se para cima.
III. Na forma interrogativa, as sobrancelhas são levantadas e há um ligeiro movimento da cabeça inclinando-se para cima e para baixo. Pode, ainda, vir também com um intensificador representado pela boca fechada com um movimento para baixo.
Está CORRETO o que se afirma:
( ) Aos poucos, a língua de sinais vem adquirindo notoriedade e superando o estereótipo que lhe fora concedido. A escrita responde a uma demanda comunicativa alfabética, enquanto a língua de sinais atende a uma demanda específica visual e espacial. Enquanto isso, na língua oral (tanto para a fala quanto para a escrita) há a emissão de um som/palavra após o outro (linearidade); na língua sinalizada, a simultaneidade é característica recorrente devido ao uso, concomitante, da expressão, da configuração de mão e de outros itens lexicais.
( ) O acesso tardio à Libras ocorre ainda pelo desconhecimento da língua sinalizada por parte da família, pela resistência da equipe médica que orienta a família ou da própria família em entender a surdez e sua consequência linguística. Reitera-se assim que a defesa pela normalização do sujeito surdo, de modo a enquadrálo ao uso da LP falada, fez com que se disseminasse uma visão deturpada da língua sinalizada, mesmo ela ainda não podendo ser considerada como uma língua.
Mark the CORRECT item to fill in the blank.
The director, ______ film won several awards, will be speaking at the conference.
Regarding prepositions, number the second column according to the first one so as to fill in the blanks, and then mark the item that corresponds to the CORRECT sequence.
(1) on
(2) at
(3) in
( ) The day was perfect yesterday. There wasn’t a single cloud ___ the sky!
( ) The game is ___ Friday.
( ) I usually get up ____ half past eight.
The mysterious death of Alexander the Great
When Alexander the Great’s body seemingly remained unchanged for six days after his death in 323 BCE, his contemporaries could offer only one explanation. Alexander must have been a god. So… was he?
Alexander the Great first fell ill during a days-long series of parties, during one of which he collapsed, complaining of a searing pain in his back. After 10 days of intense fever, Alexander’s soldiers were brought in to see him one final time. As reported by the historian Arrian, at that point the king “could no longer speak… but he struggled to raise his head and gave each man a greeting with his eyes.”
When Alexander was declared dead on June 13, theories began forming. Had he been poisoned? Sabotaged? Had he been killed by drinking too much wine? Today we have an explanation for Alexander’s death and his period of bodily freshness that relies less on the supernatural and more on science. In 2018 Dr. Katherine Hall, a lecturer in New Zealand, proposed that Alexander the Great had Guillain-Barré syndrome, an acute autoimmune condition that results in muscle paralysis. In other words, Alexander may have been alive when he was declared dead—a mistake that could have been made when physicians mistook the shallow breathing of a coma patient for no breathing at all. If this was the case, Alexander may have been effectively murdered during embalming—a process that would have seen him disemboweled.
While we can’t travel back in time to confirm Hall’s theory, it is the only one that takes into account all the details of Alexander’s death—and his body’s mysterious life.
Encyclopaedia Britannica. Adaptation
The mysterious death of Alexander the Great
When Alexander the Great’s body seemingly remained unchanged for six days after his death in 323 BCE, his contemporaries could offer only one explanation. Alexander must have been a god. So… was he?
Alexander the Great first fell ill during a days-long series of parties, during one of which he collapsed, complaining of a searing pain in his back. After 10 days of intense fever, Alexander’s soldiers were brought in to see him one final time. As reported by the historian Arrian, at that point the king “could no longer speak… but he struggled to raise his head and gave each man a greeting with his eyes.”
When Alexander was declared dead on June 13, theories began forming. Had he been poisoned? Sabotaged? Had he been killed by drinking too much wine? Today we have an explanation for Alexander’s death and his period of bodily freshness that relies less on the supernatural and more on science. In 2018 Dr. Katherine Hall, a lecturer in New Zealand, proposed that Alexander the Great had Guillain-Barré syndrome, an acute autoimmune condition that results in muscle paralysis. In other words, Alexander may have been alive when he was declared dead—a mistake that could have been made when physicians mistook the shallow breathing of a coma patient for no breathing at all. If this was the case, Alexander may have been effectively murdered during embalming—a process that would have seen him disemboweled.
While we can’t travel back in time to confirm Hall’s theory, it is the only one that takes into account all the details of Alexander’s death—and his body’s mysterious life.
Encyclopaedia Britannica. Adaptation
The tense and aspect of the underlined verbs below are:
Alexander the Great first fell ill during a days-long series of parties, during one of which he collapsed, complaining of a searing pain in his back. After 10 days of intense fever, Alexander’s soldiers were brought in to see him one final time. As reported by the historian Arrian, at that point the king “could no longer speak… but he struggled to raise his head and gave each man a greeting with his eyes.”
The mysterious death of Alexander the Great
When Alexander the Great’s body seemingly remained unchanged for six days after his death in 323 BCE, his contemporaries could offer only one explanation. Alexander must have been a god. So… was he?
Alexander the Great first fell ill during a days-long series of parties, during one of which he collapsed, complaining of a searing pain in his back. After 10 days of intense fever, Alexander’s soldiers were brought in to see him one final time. As reported by the historian Arrian, at that point the king “could no longer speak… but he struggled to raise his head and gave each man a greeting with his eyes.”
When Alexander was declared dead on June 13, theories began forming. Had he been poisoned? Sabotaged? Had he been killed by drinking too much wine? Today we have an explanation for Alexander’s death and his period of bodily freshness that relies less on the supernatural and more on science. In 2018 Dr. Katherine Hall, a lecturer in New Zealand, proposed that Alexander the Great had Guillain-Barré syndrome, an acute autoimmune condition that results in muscle paralysis. In other words, Alexander may have been alive when he was declared dead—a mistake that could have been made when physicians mistook the shallow breathing of a coma patient for no breathing at all. If this was the case, Alexander may have been effectively murdered during embalming—a process that would have seen him disemboweled.
While we can’t travel back in time to confirm Hall’s theory, it is the only one that takes into account all the details of Alexander’s death—and his body’s mysterious life.
Encyclopaedia Britannica. Adaptation
A respeito das doenças do sistema genitourinário na população pediátrica, avaliar se as afirmativas são certas (C) ou erradas (E) e assinalar a sequência correspondente.
( ) Nos lactentes do sexo feminino, as válvulas uretrais posteriores constituem a causa mais comum de obstrução da via de saída da bexiga.
( ) A agenesia renal unilateral pode ocorrer como anormalidade isolada ou como componente de distúrbios de síndrome, como a síndrome de Turner.
( ) O desenvolvimento do rim metanéfrico começa caudalmente no embrião e, com nove semanas de gestação, o rim já atingiu seu nível normal.
A lesão renal aguda (LRA) é definida como diminuição abrupta (horas a dias) da taxa de filtração glomerular (TFG), resultando em acúmulo de produtos nitrogenados e desequilíbrio hidroeletrolítico. Considerando-se a classificação das causas da LRA conforme seu mecanismo, relacionar as colunas e assinalar a sequência correspondente.
(1) Pré-renal.
(2) Renal intrínseca.
(3) Pós-renal.
( ) Ocorre por obstrução do trato urinário - ureteral bilateral (ou unilateral em rim único), vesical ou uretral. Uma causa comum em neonatos é a válvula de uretra posterior.
( ) Ocorre por dano direto ao parênquima renal.
( ) Resulta da depleção de volume circulante absoluto (p. ex., desidratação) ou relativo (p. ex., choque séptico, insuficiência cardíaca), com consequente diminuição da perfusão renal.
( ) Há a ativação de mecanismos compensatórios em uma tentativa de elevar a perfusão renal, com aumento do tônus simpático, produção de prostaglandinas renais e ativação do eixo renina-angiotensina.
Muitos fármacos necessitam sofrer biotransformação em formas mais polares para que sejam eliminados do corpo. As reações de biotransformação são designadas como reações da fase I, que tornam o agente mais polar através de oxidação, redução ou hidrólise. Em termos quantitativos, as enzimas da fase I de maior importância são os citocromos P450, uma superfamília de proteínas contendo heme, que catalisam o metabolismo de muitas substâncias lipofílicas. As isozimas do citocromo P450 denominam-se CYP e são agrupadas pelo grau de identidade na sua sequência de aminoácidos. Nesse contexto, assinalar a alternativa que se refere ao padrão de desenvolvimento para enzimas citocromo P450 importantes no recém-nascido:
Não aparentes no fígado fetal. Dados inferenciais, utilizando o processamento da fenitoína como sonda farmacológica inespecífica, sugerem atividade baixa durante a primeira semana devida, sendo a atividade de adulto alcançada até os 6 meses de idade, com atividade máxima até os 3 a 4 anos de idade. Metabolismo induzido por rifampicina e fenobarbital e inibido por cimetidina.
A mudança dos hábitos alimentares e de vida e o aumento do sedentarismo, nas últimas décadas, têm favorecido o excesso de peso, que está associado ao aumento da morbidade e à piora da qualidade de vida desde a infância, além de aumento da mortalidade futura. Sobre a obesidade endógena, analisar os itens.
I. O diagnóstico da obesidade endógena deve ser suspeitado, especialmente, naquelas crianças que já apresentam importante ganho de peso nos primeiros anos de vida.
II. Aspectos específicos na anamnese/exame físico ajudam na suspeição de causas genéticas: grupo de sinais relacionado a síndromes conhecidas, presença de deficiência intelectual ou atraso do desenvolvimento neuropsicomotor, desaceleração no crescimento linear, comprometimento prévio do sistema nervoso central.
III. A avaliação do IMC [peso (kg)/estatura (m2)] comparada aos padrões para idade e sexo é adequada para definir o excesso de peso.
Está CORRETO o que se afirma:
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